max in WT synaptoneurosomes, suggesting that Src signaling may be downregulated in KI synapses. However, our ability to rescue SERT function in KI midbrain synaptoneurosomes from the inhibition of FAK signifies elevated FAK signaling downstream from the Pro32Pro33 mutant, as verified by improved pFAK localization in 5-HT synapses. Our information c